Low-fecundity dhc-1; mel-28 C. elegans mutants do not have gonad mitosis defects

2024/12/06 by Julia Stobierska, Anita Fernandez
Medicine · #Assisted Reproductive Technology and Twin Pregnancy #Reproductive Health and Technologies

paper · doi:10.17912/micropub.biology.001445

openalex publication_date 2024/12/06 · openalex created_date 2025/02/07 · openalex updated_date 2026/07/01

Abstract

double mutants is not caused by a mitotically-inactive gonad and instead has a different and yet-to-be-determined basis.

Related

• Sterility in the offspring of spr-5; met-2 mutants may be caused by inherited H3K4 methylation and altered germline transcription
• Conditional Changes in Brood Size and Speed of Development After Long-term Laboratory Culturing of C. elegans Wild-type Strains
• Loss of the vitellogenins confers a fitness disadvantage but does not impact brood size in C. elegans
• Sexually dimorphic control of crossover distribution by the conserved ATPase PCH-2 in C. elegans
• Identification of a new allele of catp-4