Gene-Gene and Gene-Environment Interactions Involving HLA-DRB1, PTPN22, and Smoking in Two Subsets of Rheumatoid Arthritis

2007/04/11 by Henrik Källberg, Leonid Padyukov, Robert M. Plenge +10 · 5 citations
Biochemistry, Genetics and Molecular Biology · Medicine · #Cytokine Signaling Pathways and Interactions #Diabetes and associated disorders #Rheumatoid Arthritis Research and Therapies

paper · pdf · doi:10.1086/516736

openalex publication_date 2007/04/11 · crossref created 2007/04/11 · crossref issued 2007/05/01 · crossref published 2007/05/01 · crossref published-print 2007/05/01 · openalex created_date 2025/10/10 · crossref deposited 2025/11/02 · crossref indexed 2026/07/02 · openalex updated_date 2026/08/01

Citations

• Calculating measures of biological interaction
• A new model for an etiology of rheumatoid arthritis: Smoking may trigger HLA–DR (shared epitope)–restricted immune reactions to autoantigens modified by citrullination
• The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis

Cited by

• Immunity to Citrullinated Proteins in Rheumatoid Arthritis
• Specific interaction between genotype, smoking and autoimmunity to citrullinated α-enolase in the etiology of rheumatoid arthritis
• Rheumatoid arthritis
• Anti-CCP antibodies: the past, the present and the future
• Lars Klareskog [wikipedia]