Specific interaction between genotype, smoking and autoimmunity to citrullinated α-enolase in the etiology of rheumatoid arthritis

2009/11/08 by Hiba Mahdi, Benjamin A Fisher, Benjamin A. Fisher +14 · 2 citations
Immunology and Microbiology · Medicine · #Rheumatoid Arthritis Research and Therapies #Toxin Mechanisms and Immunotoxins #Viral Infections and Immunology Research

paper · doi:10.1038/ng.480

crossref issued 2009/11/08 · crossref published 2009/11/08 · crossref published-online 2009/11/08 · openalex publication_date 2009/11/08 · crossref created 2009/11/08 · crossref published-print 2009/12/01 · openalex created_date 2016/06/24 · crossref deposited 2024/03/17 · crossref indexed 2026/06/02 · openalex updated_date 2026/08/01

Citations

• Calculating measures of biological interaction
• The diagnostic properties of rheumatoid arthritis antibodies recognizing a cyclic citrullinated peptide
• A new model for an etiology of rheumatoid arthritis: Smoking may trigger HLA–DR (shared epitope)–restricted immune reactions to autoantigens modified by citrullination
• Gene-Gene and Gene-Environment Interactions Involving HLA-DRB1, PTPN22, and Smoking in Two Subsets of Rheumatoid Arthritis
• Immunity to Citrullinated Proteins in Rheumatoid Arthritis
• The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis

Cited by

• Anti-CCP antibodies: the past, the present and the future
• Lars Klareskog [wikipedia]