Expression of the gap-junction connexins 26 and 30 in the rat cochlea

1998/11/03 by J. Lautermann, Wouter-Jan F. ten Cate, Wouter-Jan Frederik ten Cate +7 · 239 citations
Biochemistry, Genetics and Molecular Biology · Neuroscience · #Anatomy #Biochemistry #Biology #Cell biology #Cochlea #Connexin #Connexins and lens biology #Gap junction #Gene #Hearing, Cochlea, Tinnitus, Genetics #Inner ear #Intracellular #Ion channel regulation and function #Organ of Corti #Spiral ganglion #Spiral ligament #Western blot

paper · doi:10.1007/s004410051192

published in Cell and Tissue Research 294(3), 415-420 (Springer Science and Business Media LLC)

crossref issued 1998/11/03 · crossref published 1998/11/03 · crossref published-print 1998/11/03 · openalex publication_date 1998/11/03 · crossref created 2002/08/25 · crossref deposited 2019/05/29 · openalex created_date 2025/10/10 · crossref indexed 2026/05/27 · openalex updated_date 2026/07/30

Citations

• Connexin 26 mutations in hereditary non-syndromic sensorineural deafness
• Gap junctions in the rat cochlea: immunohistochemical and ultrastructural analysis

Cited by

• A deletion mutation in GJB6 cooperating with a GJB2 mutation in trans in non-syndromic deafness: A novel founder mutation in Ashkenazi Jews
• Gap Junctions and Cochlear Homeostasis
• Human Connexin 30 (GJB6), a Candidate Gene for Nonsyndromic Hearing Loss: Molecular Cloning, Tissue-Specific Expression, and Assignment to Chromosome 13q12
• Mutations in GJB6 cause nonsyndromic autosomal dominant deafness at DFNA3 locus
• A Deletion Involving the Connexin 30 Gene in Nonsyndromic Hearing Impairment
• Connexin30 (Gjb6)-deficiency causes severe hearing impairment and lack of endocochlear potential
• A large deletion including most of GJB6 in recessive non syndromic deafness: a digenic effect?
• Coordinated control of connexin 26 and connexin 30 at the regulatory and functional level in the inner ear
• Human connexin26 and connexin30 form functional heteromeric and heterotypic channels
• GJB6 [wikipedia]