Effects of mitochondrial complex I subunit NDUFS5 deficiency on osteogenic differentiation

2026/07/28 by Junyan Peng, Qian Zhang, Xingyu Zhang +5

paper · doi:10.1016/j.jdent.2026.106935

crossref created 2026/07/28 · crossref deposited 2026/08/07 · crossref indexed 2026/08/07 · crossref issued 2026/12/01 · crossref published 2026/12/01 · crossref published-print 2026/12/01

Citations

• The multifaceted roles of mitochondria in osteoblasts: from energy production to mitochondrial-derived vesicle secretion
• AIFM1 is a component of the mitochondrial disulfide relay that drives complex I assembly through efficient import of NDUFS5
• Osteoblast–osteoclast interactions
• Oxidation of NADH and ROS production by respiratory complex I
• Fis1, Mff, MiD49, and MiD51 mediate Drp1 recruitment in mitochondrial fission
• The human NADH:ubiquinone oxidoreductase NDUFS5 (15kDa) subunit: cDNA cloning, chromosomal localization, tissue distribution and the absence of mutations in isolated complex I‐deficient patients
• The operation of the γ-aminobutyrate bypath of the tricarboxylic acid cycle in brain tissue in vitro