Early sympathetic islet neuropathy in autoimmune diabetes: lessons learned and opportunities for investigation

2016/06/24 by Thomas O. Mundinger, Gerald J. Taborsky · 1 citation
Biochemistry, Genetics and Molecular Biology · Medicine · Neuroscience · #Biology #Diabetes Management and Research #Diabetes and associated disorders #Diabetes mellitus #Endocrinology #Immune system #Immunology #Insulitis #Internal medicine #Islet #Medicine #Neuroscience #Pancreatic function and diabetes #Type 1 diabetes

paper · pdf · doi:10.1007/s00125-016-4026-0

crossref issued 2016/06/24 · crossref published 2016/06/24 · crossref published-online 2016/06/24 · openalex publication_date 2016/06/24 · crossref created 2016/06/25 · crossref published-print 2016/10/01 · crossref deposited 2022/07/02 · openalex created_date 2025/10/10 · crossref indexed 2026/01/16 · openalex updated_date 2026/08/01

Citations

• Intraislet Hyperinsulinemia Prevents the Glucagon Response to Hypoglycemia Despite an Intact Autonomic Response
• Human Type 1 Diabetes Is Characterized by an Early, Marked, Sustained, and Islet-Selective Loss of Sympathetic Nerves
• The Effect of Intensive Treatment of Diabetes on the Development and Progression of Long-Term Complications in Insulin-Dependent Diabetes Mellitus

Cited by

• Type 1 diabetes [wikipedia]