Excess biglycan causes eyelid malformation by perturbing muscle development and TGF-α signaling

2004/10/12 by Yasuhito Hayashi, Chia‐Yang Liu, Chia-Yang Liu +12 · 13 citations
Biochemistry, Genetics and Molecular Biology · Medicine · #Proteoglycans and glycosaminoglycans research #Corneal Surgery and Treatments #Fibroblast Growth Factor Research

paper · doi:10.1016/j.ydbio.2004.09.022

Cited by

• Biglycan in the Skeleton
• Biglycan regulates the expression and sarcolemmal localization of dystrobrevin, syntrophin, and nNOS. [europepmc]
• Changes in gene expression in human meibomian gland dysfunction. [europepmc]
• A mouse model of spontaneous preterm birth based on the genetic ablation of biglycan and decorin. [europepmc]
• Wakayama symposium: challenges of future research in ocular surface cell biology. [europepmc]
• Perturbed meibomian gland and tarsal plate morphogenesis by excess TGFα in eyelid stroma. [europepmc]
• Meibomian gland morphogenesis requires developmental eyelid closure and lid fusion. [europepmc]
• Role of EGF receptor signaling on morphogenesis of eyelid and meibomian glands. [europepmc]
• Biglycan in the Skeleton. [europepmc]
• Roles of Two Small Leucine-Rich Proteoglycans Decorin and Biglycan in Pregnancy and Pregnancy-Associated Diseases. [europepmc]
• A Systematic Approach of the Intrauterine Morphogenesis of the Human Palpebral Apparatus. [europepmc]
• Small leucine rich proteoglycans: Biology, function and their therapeutic potential in the ocular surface. [europepmc]
• The Germinal Origin of Salivary and Lacrimal Glands and the Contributions of Neural Crest Cell-Derived Epithelium to Tissue Regeneration. [europepmc]