The Rho Kinases: Critical Mediators of Multiple Profibrotic Processes and Rational Targets for New Therapies for Pulmonary Fibrosis

2014/11/14 by Rachel S. Knipe, Andrew M. Tager, James K. Liao
Medicine · Biochemistry, Genetics and Molecular Biology · #Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis #Fibroblast Growth Factor Research #Lung Cancer Treatments and Mutations

paper · pdf · doi:10.1124/pr.114.009381

Citations

• Making sense of latent TGFβ activation
• Phosphorylation of CPI‐17, an inhibitory phosphoprotein of smooth muscle myosin phosphatase, by Rho‐kinase
• Comparison of the morphological and biochemical changes in normal human lung fibroblasts and fibroblasts derived from lungs of patients with idiopathic pulmonary fibrosis during FasL‐induced apoptosis