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Elements of a theory for the mechanisms controlling abundance, diversity, and biogeochemical role of lytic bacterial viruses in aquatic systems

2000/09/01 by T. Frede Thingstad · 1,058 citations
Biochemistry, Genetics and Molecular Biology · Environmental Science · #Abundance (ecology) #Bacteria #Bacterial cell structure #Bacterial virus #Bacteriophage #Bacteriophages and microbial interactions #Biochemistry #Biogeochemical cycle #Biology #Ecology #Escherichia coli #Evolution and Genetic Dynamics #Genetics #Lysis #Lytic cycle #Microbial Community Ecology and Physiology #Virology #Virus

paper · doi:10.4319/lo.2000.45.6.1320

published in Limnology and Oceanography 45(6), 1320-1328 (Wiley)

openalex publication_date 2000/09/01 · openalex created_date 2025/10/10 · openalex updated_date 2026/07/31

Abstract

Mechanisms controlling virus abundance and partitioning of loss of bacterial production between viral lysis and protozoan predation are discussed within the framework of an idealized Lotka‐Volterra–type model. This combines nonselective protozoan predation with host‐selective viral lysis of bacteria. The analysis leads to a reciprocal relationship between bacterial diversity and viruses, in which coexistence of competing bacterial species is ensured by the presence of viruses that ‘kill the winner,’ whereas the differences in substrate affinity between the coexisting bacterial species determine viral abundance. The ability of the model to reproduce published observations, such as an approximate 1:10 ratio between bacterial and viral abundance, and the ability of viral lysis to account for 10–50% of bacterial loss are discussed.

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