IL-1 and autoinflammatory disease: biology, pathogenesis and therapeutic targeting

2022/06/21 by Lori Broderick, Hal M. Hoffman · 1 voice · 1 citation
Biochemistry, Genetics and Molecular Biology · Immunology and Microbiology · #Biomarkers in Disease Mechanisms #IL-33, ST2, and ILC Pathways #Inflammasome and immune disorders

paper · pdf · doi:10.1038/s41584-022-00797-1

crossref issued 2022/06/21 · crossref published 2022/06/21 · crossref published-online 2022/06/21 · openalex publication_date 2022/06/21 · crossref created 2022/06/21 · crossref published-print 2022/08/01 · crossref deposited 2024/12/11 · openalex created_date 2025/10/10 · crossref indexed 2026/07/29 · openalex updated_date 2026/07/29

Citations

• Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)
• Function and regulation of IL‐1α in inflammatory diseases and cancer

Cited by

• Sex Differences in Circulating Inflammatory, Immune, and Tissue Growth Markers Associated with Fabry Disease-Related Cardiomyopathy

Discussions

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