Single-cell multi-omic and spatial profiling of human kidneys implicates the fibrotic microenvironment in kidney disease progression

2024/07/24 by Amin Abedini, Jonathan Levinsohn, Konstantin A. Klötzer +27 · 7 citations

paper · doi:10.1038/s41588-024-01802-x

Cited by

• Shared mechanisms of organ fibrosis
• A comprehensive benchmarking for spatially resolved transcriptomics clustering methods across variable technologies, organs, and replicates
• Exome-Wide Analysis Identifies a Rare EXD3 Missense Variant Associated With Diabetic Kidney Disease
• Critical role of transcription factor SOX4 in tubular epithelial cell dedifferentiation and fibroblast activation during kidney fibrosis
• Tubular cell-specific platelet-derived growth factor-B drives formation of fibrogenic niches in kidney fibrosis
• When imaging technology meets single-cell omics: new paradigm in developmental biology
• Advances in computational nephropathology